Митху Сторони - Без стресса. Научный подход к борьбе с депрессией, тревожностью и выгоранием

M. Kawano, M. Miyoshi, A. Ogawa, F. Sakai, and Y. Kadooka, « Lactobacillus gasseri SBT2055 Inhibits Adipose Tissue Inflammation and Intestinal Permeability in Mice Fed a High-fat Diet». Journal of Nutritional Science 5 (2016): e23, doi: 10.1017/jns.2016.12.

J. P. Després and I. Lemieux, «Abdominal Obesity and Metabolic Syndrome». Nature 444, no. 7121 (Dec. 2006): 881–887.

Биоимпедансометрия – метод оценки состава тела и в частности количества жира в организме.

L. E. Gyllenhammer, M. J. Weigensberg, D. Spruijt-Metz, H. Allayee, M. I. Goran, and J. N. Davis, «Modifying Influence of Dietary Sugar in the Relationship between Cortisol and Visceral Adipose Tissue in Minority Youth». Obesity 22, no. 2 (2014): 474–481.

H. R. Hong, J. O. Jeong, J. Y. Kong, S. H. Lee, S. H. Yang, C. D. Ha, and H. S. Kang, «Effect of Walking Exercise on Abdominal Fat, Insulin Resistance and Serum Cytokines in Obese Women». Journal of Exercise Nutrition & Biochemistry 18, no. 3 (Sept. 2014): 277–285.

R. Ross, R. Hudson, P. J. Stotz, and M. Lam, «Effects of Exercise Amount and Intensity on Abdominal Obesity and Glucose Tolerance in Obese Adults: A Randomized Trial». Annals of Internal Medicine 162, no. 5 (Mar. 2015): 325–334.

S. E. Keating, D. A. Hackett, H. M. Parker, H. T. O’Connor, J. A. Gerofi, A. Sainsbury, M. K. Baker, V. H. Chuter, I. D. Caterson, J. George, and N. A. Johnson, «Effect of Aerobic Exercise Training Dose on Liver Fat and Visceral Adiposity». Journal of Hepatology 63, no. 1 (Jul. 2015): 174–182.

G. R. Logan, N. Harris, S. Duncan, L. D. Plank, F. Merien, and G. Schofield, «Low-active Male Adolescents: A Dose Response to High-intensity Interval Training». Medicine & Science in Sports & Exercise 48, no. 3 (Mar. 2016): 481–490.

K. E. Koopman, M. W. Caan, A. J. Nederveen, et al., «Hypercaloric Diets with Increased Meal Frequency, but not Meal Size, Increase Intrahepatic Triglycerides: A Randomized Controlled Trial». Hepatology 60, no. 2 (2014): 545–553.

M. Gleeson, N. C. Bishop, D. J. Stensel, et al., «The Anti-inflammatory Effects of Exercise: Mechanisms and Implications for the Prevention and Treatment of Disease». Nature Reviews Immunology 11 (2011): 607–615.

G. I. Lancaster and M. A. Febbraio, «The Immunomodulating Role of Exercise in Metabolic Disease». Trends in Immunology (2014): 262–269.

C. Kasapis and P. D. Thompson, «The Effects of Physical Activity on Serum C-reactive Protein and Inflammatory Markers: A Systematic Review». Journal of the American College of Cardiology 45 (2005): 1563–1569.

A. Masuda, M. Nakazato, T. Kihara, S. Minagoe, and C. Tei, «Repeated Thermal Therapy Diminishes Appetite Loss and Subjective Complaints in Mildly Depressed Patients». Psychosomatic Medicine 67, no. 4 (Jul. – Aug. 2005): 643–647.

C. W. Janssen, C. A. Lowry, M. R. Mehl, J. J. Allen, K. L. Kelly, D. E. Gartner, A. Medrano, T. K. Begay, K. Rentscher, J. J. White, A. Fridman, L. J. Roberts, M. L. Robbins, K. U. Hanusch, S. P. Cole, and C. L. Raison, «Whole-body Hyperthermia for the Treatment of Major Depressive Disorder: A Randomized Clinical Trial». JAMA Psychiatry 73, no. 8 (Aug. 2016): 789–795.

M. Bauer, S. Berman, T. Stamm, M. Plotkin, M. Adli, M. Pilhatsch, E. D. London, G. S. Hellemann, P. C. Whybrow, and F. Schlagenhauf, «Levothyroxine Effects on Depressive Symptoms and Limbic Glucose Metabolism in Bipolar Disorder: A Randomized, Placebocontrolled Positron Emission Tomography Study». Molecular Psychiatry 21, no. 2 (Feb. 2016): 229–236.

M. Krause, M. S. Ludwig, T. G. Heck, and H. K. Takahashi, «Heat Shock Proteins and Heat Therapy for Type 2 Diabetes: Pros and Cons». Current Opinion in Clinical Nutrition and Metabolic Care 18, no. 4 (Jul. 2015): 374–380.

T. Laukkanen, S. Kunutsor, J. Kauhanen, and J. A. Laukkanen, «Sauna Bathing is Inversely Associated with Dementia and Alzheimer’s Disease in Middle-aged Finnish Men». Age and Ageing 1, no. 5 (Dec. 2016) ( epub ahead of print ) doi: 10.1093/ageing/afw212.

P. Cassano, S. R. Petrie, M. R. Hamblin, T. A. Henderson, and D. V. Iosifescu, «Review of Transcranial Photobiomodulation for Major Depressive Disorder: Targeting Brain Metabolism, Inflammation, Oxidative Stress, and Neurogenesis». Neurophotonics 3, no. 3 (Jul. 2016): 031404.

L. Detaboada et al., «Transcranial Application of Low-energy Laser Irradiation Improves Neurological Deficits in Rats Following Acute Stroke». Lasers in Surgery and Medicine 38, no. 1 (2006): 70–73.

H. Araki et al., «Reduction of Interleukin-6 Expression in Human Synoviocytes and Rheumatoid Arthritis Rat Joints by Linear Polarized Near Infrared Light (Superlizer) Irradiation». Laser Therapy 20, no. 4 (2011): 293.

M. Yamaura et al., «Low Level Light Effects on Inflammatory Cytokine Production by Rheumatoid Arthritis Synoviocytes». Lasers in Surgery and Medicine 41, no. 4 (2009): 282–290.

J. Khuman et al., «Low-level Laser Light Therapy Improves Cognitive Deficits and Inhibits Microglial Activation after Controlled Cortical Impact in Mice». Journal of Neurotrauma 29, no. 2 (2012): 408–417.

J. C. Rojas, A. K. Bruchey, and F. Gonzalez-Lima, «Low-level Light Therapy Improves Cortical Metabolic Capacity and Memory Retention». Journal of Alzheimer’s Disease 32, no. 3 (2012): 741–752.

D. Barrett and F. Gonzalez-Lima, «Transcranial Infrared Laser Stimulation Produces Beneficial Cognitive and Emotional Effects in Humans». Neuroscience 230 (2013): 13–23.

Гидрофобность – свойство молекул «стремиться» избежать контакта с водой.

X. Qin and E. A. Deitch, «Dissolution of Lipids from Mucus: A Possible Mechanism for Prompt Disruption of Gut Barrier Function by Alcohol». Toxicology Letters 232, no. 2 (Jan. 2015): 356–362.

J. Connor, «Alcohol Consumption as a Cause of Cancer». Addiction 112, no. 2 (Feb. 2017): 222–228.

R. Nirupama, M. Devaki, and H. N. Yajurvedi, «Chronic Stress and Carbohydrate Metabolism: Persistent Changes and Slow Return to Normalcy in Male Albino Rats». Stress 15, no. 3 (May 2012): 262–271.

S. Garbarino and N. Magnavita, «Work Stress and Metabolic Syndrome in Police Officers. A Prospective Study». ed. V. Grolmusz, PLoS ONE10 , no. 12 (2015): e0144318.

A. Hino, A. Inoue, K. Mafune, T. Nakagawa, T. Hayashi, and H. Hiro, «Changes in the Psychosocial Work Characteristics and Insulin Resistance among Japanese Male Workers: A Three-year Follow-up Study». Journal of Occupational Health 58, no. 6 (Nov. 2016): 543–562.

B. Schmidt, J. A. Bosch, M. N. Jarczok, R. M. Herr, A. Loerbroks, E. van Vianen, and J. E. Fischer, «Effort-reward Imbalance Is Associated with the Metabolic Syndrome – Findings from the Mannheim Industrial Cohort Study (MICS)». International Journal of Cardiology 178 (Jan. 2015): 24–28.

T. Almadi, I. Cathers, and C. M. Chow, «Associations among Workrelated Stress, Cortisol, Inflammation, and Metabolic Syndrome». Psychophysiology 50, no. 9 (Sept. 2013): 821–830.

V. K. Tsenkova, D. Carr, C. L. Coe, and C. D. Ryff, «Anger, Adiposity, and Glucose Control in Nondiabetic Adults: Findings from Midus II». Journal of Behavioral Medicine 37, no. 1 (2014): 37–46.

K. Räikkönen, L. Keltikangas-Järvinen, and A. Hautanen, «The Role of Psychological Coronary Risk Factors in Insulin and Glucose Metabolism». Journal of Psychosomatic Research 38, no. 7 (Oct. 1994): 705–713.

S. H. Boyle, A. Georgiades, B. H. Brummett, et al., «Associations between Central Nervous System Serotonin, Fasting Glucose and Hostility in African American Females». Annals of Behavioral Medicine: A Publication of the Society of Behavioral Medicine 49, no. 1 (2015): 49–57.

A. Quincozes-Santos, L. D. Bobermin, A. M. de Assis, C. A. Gonçalves, and D. O. Souza, «Fluctuations in Glucose Levels Induce Glial Toxicity with Glutamatergic, Oxidative and Inflammatory Implications». Biochimica et Biophysica Acta 1863, no. 1 (Jan. 2017): 1–14.

D. E. Rivera-Aponte, M. P. Méndez-González, A. F. Rivera-Pagán, Y. V. Kucheryavykh, L. Y. Kucheryavykh, S. N. Skatchkov, and M. J. Eaton, «Hyperglycemia Reduces Functional Expression of Astrocytic Kir4.1 channels and Glial Glutamate Uptake». Neuroscience 310 (Dec. 2015): 216–223.

When one brain cell passes a message to another and wants the recipient to become activated or «excited». it sends it the chemical messenger glutamate. Glutamate will keep exciting the recipient brain until it is cleared away. If it is not cleared away, the brain cell can die of too much excitation or «excitotoxicity.» Elevated blood sugar, at levels comparable to those seen in the setting of insulin resistance and diabetes, can interfere with the clearing away of glutamate.

G. B. Stefano, S. Challenger, and R. M. Kream, «Hyperglycemiaassociated Alterations in Cellular Signaling and Dysregulated Mitochondrial Bioenergetics in Human Metabolic Disorders». European Journal of Nutrition 55, no. 8 (Dec. 2016): 2339–2345.

W. Cai, J. Uribarri, L. Zhu, X. Chen, S. Swamy, Z. Zhao, F. Grosjean, C. Simonaro, G. A. Kuchel, M. Schnaider-Beeri, M. Woodward, G. E. Striker, and H. Vlassara, «Oral Glycotoxins Are a Modifiable Cause of Dementia and the Metabolic Syndrome in Mice and Humans». Proceedings of the National Academy of Sciences of the United States of America 111, no. 13 (Apr. 2014): 4940–4945.

K. A. Page, A. Williamson, N. Yu, E. C. McNay, J. Dzuira, R. J. McCrimmon, and R. S. Sherwin, «Medium-chain Fatty Acids Improve Cognitive Function in Intensively Treated Type 1 Diabetic Patients and Support in Vitro Synaptic Transmission during Acute Hypoglycemia». Diabetes 58, no. 5 (May 2009): 1237–1244.

E. Bullmore and O. Sporns, «The Economy of Brain Network Organization.» Nature Reviews Neuroscience 13 (2012): 336–349.